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Survival of Cultured Neurons from Amyloid Precursor Protein Knock-Out Mice  against Alzheimer's Amyloid-β Toxicity and Oxidative Stress | Journal of  Neuroscience
Survival of Cultured Neurons from Amyloid Precursor Protein Knock-Out Mice against Alzheimer's Amyloid-β Toxicity and Oxidative Stress | Journal of Neuroscience

IJMS | Free Full-Text | Deciphering the Potential Neuroprotective Effects  of Luteolin against Aβ1–42-Induced Alzheimer's Disease | HTML
IJMS | Free Full-Text | Deciphering the Potential Neuroprotective Effects of Luteolin against Aβ1–42-Induced Alzheimer's Disease | HTML

Deficiency of Neuronal p38α MAPK Attenuates Amyloid Pathology in Alzheimer  Disease Mouse and Cell Models through Facilitating Lysosomal Degradation of  BACE1* - Journal of Biological Chemistry
Deficiency of Neuronal p38α MAPK Attenuates Amyloid Pathology in Alzheimer Disease Mouse and Cell Models through Facilitating Lysosomal Degradation of BACE1* - Journal of Biological Chemistry

Luteolin ameliorates depression-like behaviors by suppressing ER stress in  a mouse model of Alzheimer's disease - ScienceDirect
Luteolin ameliorates depression-like behaviors by suppressing ER stress in a mouse model of Alzheimer's disease - ScienceDirect

Caffeine inhibits lipopolysaccharide (LPS)-induced oxidative stress in... |  Download Scientific Diagram
Caffeine inhibits lipopolysaccharide (LPS)-induced oxidative stress in... | Download Scientific Diagram

Copper-Targeting Approaches in Alzheimer's Disease: How To Improve the  Fallouts Obtained from in Vitro Studies | Inorganic Chemistry
Copper-Targeting Approaches in Alzheimer's Disease: How To Improve the Fallouts Obtained from in Vitro Studies | Inorganic Chemistry

Differential protein expression in diverse brain areas of Parkinson's and  Alzheimer's disease patients | Scientific Reports
Differential protein expression in diverse brain areas of Parkinson's and Alzheimer's disease patients | Scientific Reports

Biomedicines | Free Full-Text | Relationship between Cognitive Dysfunction  and Age-Related Variability in Oxidative Markers in Isolated Mitochondria  of Alzheimer’s Disease Transgenic Mouse Brains
Biomedicines | Free Full-Text | Relationship between Cognitive Dysfunction and Age-Related Variability in Oxidative Markers in Isolated Mitochondria of Alzheimer’s Disease Transgenic Mouse Brains

Vanillic acid attenuates Aβ1-42-induced oxidative stress and cognitive  impairment in mice | Scientific Reports
Vanillic acid attenuates Aβ1-42-induced oxidative stress and cognitive impairment in mice | Scientific Reports

Oxidative stress affects processing of amyloid precursor protein in  vascular endothelial cells | PLOS ONE
Oxidative stress affects processing of amyloid precursor protein in vascular endothelial cells | PLOS ONE

Epigenetic mechanisms underlying cognitive impairment and Alzheimer disease  hallmarks in 5XFAD mice | Aging
Epigenetic mechanisms underlying cognitive impairment and Alzheimer disease hallmarks in 5XFAD mice | Aging

Antioxidants | Free Full-Text | Protective Effects against the Development  of Alzheimer’s Disease in an Animal Model through Active Immunization  with Methionine-Sulfoxide Rich Protein Antigen | HTML
Antioxidants | Free Full-Text | Protective Effects against the Development of Alzheimer’s Disease in an Animal Model through Active Immunization with Methionine-Sulfoxide Rich Protein Antigen | HTML

Oxidative stress and pro‐inflammatory markers in female mice aged 2 and...  | Download Scientific Diagram
Oxidative stress and pro‐inflammatory markers in female mice aged 2 and... | Download Scientific Diagram

Downregulation of CREB expression in Alzheimer's brain and in Aβ-treated  rat hippocampal neurons | Molecular Neurodegeneration | Full Text
Downregulation of CREB expression in Alzheimer's brain and in Aβ-treated rat hippocampal neurons | Molecular Neurodegeneration | Full Text

Frontiers | The Potential of Ferroptosis-Targeting Therapies for Alzheimer's  Disease: From Mechanism to Transcriptomic Analysis
Frontiers | The Potential of Ferroptosis-Targeting Therapies for Alzheimer's Disease: From Mechanism to Transcriptomic Analysis

Ginsenoside Rg1 reduces β‑amyloid levels by inhibiting CDΚ5‑induced PPARγ  phosphorylation in a neuron model of Alzheimer's disease
Ginsenoside Rg1 reduces β‑amyloid levels by inhibiting CDΚ5‑induced PPARγ phosphorylation in a neuron model of Alzheimer's disease

Western blot analysis showing the expression levels of amyloid-protein... |  Download Scientific Diagram
Western blot analysis showing the expression levels of amyloid-protein... | Download Scientific Diagram

Markers of oxidative stress in Alzheimer's mouse brain. (A) Brain... |  Download Scientific Diagram
Markers of oxidative stress in Alzheimer's mouse brain. (A) Brain... | Download Scientific Diagram

Protective effect of paeoniflorin on inflammation and apoptosis in the  cerebral cortex of a transgenic mouse model of Alzheimer's disease
Protective effect of paeoniflorin on inflammation and apoptosis in the cerebral cortex of a transgenic mouse model of Alzheimer's disease

MKP-1 reduces Aβ generation and alleviates cognitive impairments in  Alzheimer's disease models | Signal Transduction and Targeted Therapy
MKP-1 reduces Aβ generation and alleviates cognitive impairments in Alzheimer's disease models | Signal Transduction and Targeted Therapy

Proteomic Analysis of Hippocampus in a Mouse Model of Depression Reveals  Neuroprotective Function of Ubiquitin C-terminal Hydrolase L1 (UCH-L1) via  Stress-induced Cysteine Oxidative Modifications* - Molecular & Cellular  Proteomics
Proteomic Analysis of Hippocampus in a Mouse Model of Depression Reveals Neuroprotective Function of Ubiquitin C-terminal Hydrolase L1 (UCH-L1) via Stress-induced Cysteine Oxidative Modifications* - Molecular & Cellular Proteomics

A third-generation mouse model of Alzheimer's disease shows early and  increased cored plaque pathology composed of wild-type human amyloid β  peptide - Journal of Biological Chemistry
A third-generation mouse model of Alzheimer's disease shows early and increased cored plaque pathology composed of wild-type human amyloid β peptide - Journal of Biological Chemistry